Buy Zydena (Udenafil) online, Dong-A Pharmaceutical Co., Korea

Dong-A Pharmaceutical Co. (Korea)
4 Tabs x 100mg Total 400mg

Zydena (Udenafil)

Active Ingredient: Udenafil

Udenafil– trade name «Zydena ®» is one of the four molecules investigated for the treatment of erectile dysfunction with proven clinical efficacy, high safety and acceptability to the patient. The unique qualities of Zydena distinguish it from commercially available inhibitors of other members of the group focfodiesterazy fifth type.

Zydena enjoy rapid and predictable onset of action, duration of up to 24 hours, high in the class of PDE-5 inhibitors, the safety profile, Zydena not have testicular toxicity. Certainly, all of these properties will be the best in its segment.

The composition and the form of Udenafil

Tablets, film-coated light-pink color with light-yellow shade, oval shaped, imprinted with the symbol “100” on one side and symbols in the form of risk sharing letters “Z” and “Y” – to another, on a break – a white or nearly white .

1 tablet contains 100 mg udenafil;


  • lactose
  • maize starch
  • colloidal silicon dioxide
  • L-hydroxypropylcellulose
  • hydroxypropyl-LF
  • talc
  • magnesium stearate.

1 or 4 tablets per pack.

Pharmacological action of Udenafil

Zydena – a drug for treatment of erectile dysfunction. Is a reversible selective inhibitor of cGMP-specific phosphodiesterase type 5 (PDE5).

Udenafil no direct relaxing effect on isolated corpus cavernosum, but enhances sexual stimulation relaxing effect of nitric oxide by inhibiting PDE-5, responsible for the breakdown of cGMP in the cavernous body. The result is relaxation of smooth muscles of arteries and blood flow to the tissues of the penis that causes erections. The drug has no effect in the absence of sexual stimulation.

Zydena improves erection and the possibility of successful sexual intercourse. The action of the drug has an optimal duration – up to 24 h. The effect is manifested as early as 30 minutes after taking the drug in the presence of sexual arousal.

In the study udenafil, men revealed no clinically significant effect on the drug quantity and concentration of sperm motility and morphology of spermatozoa.

Udenafil Indications

Treatment of erection disorders, characterized by the inability to achieve or maintain penile erection sufficient for satisfactory sexual intercourse.

Udenafil Dosing and Administration

Pills taken by mouth, with or without food, for 30 minutes before the alleged sexual activity.

Recommended dose – 100 mg. If necessary, taking into account individual performance and portability, the dose may be increased to 200 mg. The maximum recommended frequency of application – 1 times / day.

Udenafil Side effects

  • flushing
  • dizziness, eye pain
  • increased lacrimation
  • nasal congestion
  • headache

Also noted: palpitations, prolonged erections.


Sexual activity is a potential risk for patients with cardiovascular disease, hypertension, so treatment of sexual dysfunctions, including use of Zydena, should not hold for men with heart disease, in which sexual activity is not recommended.

Patients with obstruction of blood outflow from the left ventricle (aortic stenosis) may be more sensitive to the action of vasodilators, including inhibitors of PDE. Despite the lack of clinical trials of cases of prolonged erection (longer than 4 hours) and priapism (painful erection lasting more than 6 h), such phenomena are peculiar to this class of drugs. In the event of an erection lasting more than 4 hours (regardless of pain), patients should immediately seek medical help. In the absence of timely treatment of priapism can lead to irreversible damage to the erectile tissue and erectile function.

In the absence of clinical data on the use udenafil in patients older than 71 y.o. this category not recommended for patients taking the drug. Not recommended for use Zydena in combination with other treatments for erectile dysfunction.


  • rapid and predictable onset of action
  • provides necessary to erection hardness
  • increases the axial load on the penis
  • reduces the time of the refractory period (time after ejaculation until the next erection)
  • optimal duration of up to 24 hours
  • high safety profile in the class of PDE-5 inhibitors
  • Drinking alcohol and fatty food does not affect the action of udenafil
  • You can buy Zydena (Udenafil) here

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Buy Aromasin (Exemestane) / Arimidex (Anastrozole) exedrol (exemestane) Buy Anastrozole
Aromasin [Exemestane Tablets] British Dragon (Thailand)
Powerful anti-estrogen (reduces aromatization)
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60 Tabs x 25mg Total 1500mg
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Aromasin [Exemestane Tablets] / Arimidex (Anastrozole) / Femara (Letrozole)


by Anthony Roberts — Aromasin (Exemestane) is one of those weird compounds that nobody really knows what to do with. What we generally hear about it makes it very uninteresting It’s a third generation Aromatase Inhibitor (AI) just like Arimidex (Anastrozole) and Femara (Letrozole). Both of those two drugs are very efficient at stopping the conversion of androgens into estrogen, and since we have them, why bother with Aromasin? It’s a little harder to get than the other two commonly used aromatase inhibitors, because it’s not in high demand, and there’s never been a readily apparent advantage to using it. And I mean lets face it: It’s awkward-sounding. Aromasin doesn’t have much of a ring to it, and exemestane is even worse. Arimidex has a bunch of cool abbreviations (“A-dex” or just ‘dex) and even Letrozole is just “Letro” to most people. Where’s the cool nickname for Aromasin/exemestane? A-Sin? E-Stane? It just doesn’t work. It’s the black sheep of AI‘s. And why do we even need it when we have Letrozole, which is by far the most efficient AI for stopping aromatization (the process by which your body converts testosterone into estrogen)?

Letro can reduce estrogen levels by 98% or greater; clinically a dose as low as 100 mcgs has been shown to provide maximum aromatase inhibition! So why would we need any other AI‘s? Well, first of all, estrogen is necessary for healthy joints as well as a healthy immune system. So getting rid of 98% of the estrogen in your body for an extended period of time may not be the best of ideas. This may be useful on an extreme cutting cycle, leading up to a bodybuilding contest, or if you are particularly prone to gyno, but certainly can’t be used safely for extended periods of time without compromising your joints and immune system.

So that leaves us with Arimidex, which isn’t as potent as Letrozole, but at .5mgs/day will still get rid of around half (50%) of the estrogen in your body. Problem solved, right? Use Arimidex on your typical cycles, and if you are very prone to gyno or are getting ready for a contest, use Letro.

But what about Post Cycle Therapy (PCT)?

I think at this point most people are sold on the use of Nolvadex (Tamoxifen Citrate) instead of Clomid for PCT, since both compete estrogen at the receptor site, both increase serum test levels, and both drugs may also alter blood lipid profiles favorably (6). But since 20mgs of Tamoxifen is equal to 150mgs of clomid for purposes of testosterone elevation, FSH and LH, but Tamoxifen doesn’t decrease the LH response to :LHRH:. I think most people agree to Nolvadex’s superiority for PCT.

I’ve always been in favor of using Nolvadex during PCT, along with an AI (Aromatase Inhibitor), because reducing estrogen levels has been positively correlated with an increase in testosterone so in my mind, it’s be beneficial to increase testosterone by as many mechanisms as possible while trying to recover your endogenous testosterone levels after a cycle. SO which AI do we use? Letro or A-dex? Well, why don’t we just keep using whichever one we used during the cycle, and add in some Nolvadex? Unfortunately, Nolvadex will significantly reduce the blood plasma levels of both Letrozole as well as Arimidex. So if we choose to use one of them with our Nolvadex on PCT, we’re throwing away a bit of money as the Nolvadex will be reducing their effectiveness.
This, of course, is where Aromasin comes in, at 20-25mgs/day.

Aromasin, at that dose, will raise your testosterone levels by about 60%, and also help out your free to bound testosterone ratio by lowering levels of Sex Hormone Binding Globulin (SHBG), by about 20% SHBG is that nasty enzyme that binds to testosterone and renders it useless for building muscle. But what about using it along with Nolvadex for PCT?

To understand why Aromasin may be useful in conjunction with Nolvadex while both Letro and A-dex suffer reduced effectiveness, we’ll need to first understand the differences between a Type-I and Type-II Aromatase Inhibitor. Type I inhibitors (like Aromasin) are actually steroidal compounds, while type II inhibitors (like Letro and A-dex) are non-steroidal drugs. Hence, androgenic side effects are very possible with Type-I AIs, and they should probably be avoided by women.
Of course, there are some similarities between the two types of AIs both type I & type II AI‘s mimic normal substrates (essentially androgens), allowing them to compete with the substrate for access to the binding site on the aromatase enzyme. After this binding, the next step is where things differ greatly for the two different types of AI’s. In the case of a type-I AI, the noncompetitive inhibitor will bind, and the enzyme initiates a sequence of hydroxylation; this hydroxylation produces an unbreakable covalent bond between the inhibitor and the enzyme protein. Now, enzyme activity is permanently blocked; even if all unattached inhibitor is removed. Aromatase enzyme activity can only be restored by new enzyme synthesis. Now, on the other hand, competitive inhibitors, called type II AI’s, reversibly bind to the active enzyme site, and one of two things can happen:

1.) either no enzyme activity is triggered or

2.) the enzyme is somehow triggered without effect.

The type II inhibitor can now actually disassociate from the binding site, eventually allowing renewed competition between the inhibitor and the substrate for binding to the site. This means that the effectiveness of competitive aromatase inhibitors depends on the relative concentrations and affinities of both the inhibitor and the substrate, while this is not so for noncompetitive inhibitors. Aromasin is a type-I inhibitor, meaning that once it has done its job, and deactivated the aromatase enzyme, we don’t need it anymore. Letrozole and Arimidex actually need to remain present to continue their effects. This is possibly why Nolvadex does not alter the pharmacokinetics of Aromasin.

Before we close the book on Aromasin, it’s worth noting that you can (and should) still use one of the non-steroidal AI‘s during your cycle to reduce estrogen, if necessary. When you are ready for PCT, you can then switch over to Aromasin and still experience the full effects of an AI, since there is no cross-over tolerance experienced between steroidal and non-steroidal AI‘s. Since Aromasin is about 65% efficient at suppressing estrogen, it’s certainly a very powerful agent, especially considering you won’t experience reduced effectiveness because of your concurrent use of Nolvadex or from any sort of tolerance developed by using other AI‘s on your cycle. There is also a decent amount of preclinical data suggesting that Aromasin has a beneficial effect on bone mineral metabolism that is not seen with non-steroidal agents, and it may also have beneficial effects on lipid metabolism that are not found in the non-steroidal Letro and A-dex.

Finally, as we’re going to be using Nolvadex for PCT anyway, and we ought to be using an AI with it for maximum recovery I think Aromasin- considering it’s compatibility with Nolvadex and beneficial effects on bone mineral content and lipid profile, has finally stopped being the black sheep of AIs and found a home in our Cycles.


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